The weakening of legs refers to the decrease in the power and ability of a person to move one’s legs, which can be attributed to the either the declining power of the muscles to contract or the failing of nerves signals from the brain to command the muscle to contract. In which case, any interruption of nerve signals from the brain to the limb results to leg weakness. In the case of diabetic patients, the weakening of the legs is caused by the latter or nerve damage called proximal neuropathy, which poses the risk of disability.

Diabetic neuropathies are a group of nerve disorders which is primarily caused by diabetes. This can be caused by prolonged high blood sugar levels that damage the nerve and other factors such as abnormal blood fat levels, low insulin levels, the inability of the blood vessels to bring oxygen and nutrients to nerves, nerve inflammation, nerve injuries, and even lifestyle factors like smoking and excessive alcohol drinking.

There are different kinds of diabetic neuropathies such as peripheral neuropathy (weakening of feet, toes, arms and hands), autonomic neuropathy (weakening of digestion, excretory function, and sexual response), focal neuropathy (sudden weakening one nerve or a group of nerves) and proximal neuropathy or the weakening of the legs as well as thighs, hips, or buttocks. In the case of proximal neuropathy, the weakening of the legs of diabetic patients is often not perceived empirically because it does not occur with any considerable changes in strength, mass or quality of the leg muscles. Leg weakening is primarily a result of nerve damage.

The occurrence of proximal neuropathy or the weakening of the legs among diabetics is often accompanied by symptoms such as initial numbness or tingling sensations in the legs. The risk of neuropathy is higher among patients who have diabetes for a long time, older people with diabetes as well as diabetics who are obese.

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